6 Worst Foods for Gut Health (& What to Eat Instead) background image
July 05, 2024

6 Worst Foods for Gut Health (& What to Eat Instead)

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Key Takeaways:

Key Points:

  • Unsurprisingly, the worst foods for gut health are usually the most heavily processed ones. As we nurture our microbiome, it’s important to prioritize a diet of whole and nutrient-dense foods that provide us with the tools we need to fuel our gut ecosystems.
  • People with gut dysbiosis should recognize that some healthy foods (high FODMAP foods and fiber, for example) might temporarily be problematic as they work to rebuild gut health. In this case, give yourself permission to do what you can and take recovery slowly.
  • Replace the worst foods for your gut with the best foods. Our rule of thumb is to always find the freshest and least processed foods available (yes, this even includes raw dairy).

Hippocrates famously said, “All disease begins in the gut.”

Over two thousand years later, we’re only just catching onto his ancient wisdom.

The gut is home to an entire ecosystem of bacteria, yeast, fungus, and other microbes. When in perfect balance, this ecosystem tackles so much more than just digestion.

You can thank your gut for… 

  • Blood sugar balance
  • Immune system regulation
  • Serotonin production
  • Nutrient metabolism and absorption
  • Nervous system health
  • Mood stability
  • Weight management

… and many more benefits that science is still working to uncover.

This is why it’s so important to evaluate the foods we eat regularly — and how they could positively or negatively impact our gut health.

Let’s explore the six worst foods for gut health (and what to eat instead).

Table of Contents:

6 Worst Foods for Gut Health

People with a sensitive gut know all too well that some foods are better for the digestive system than others. Let’s unpack some of the worst foods for gut health (particularly, the microbiome) and find some better alternatives.

1. Chemically Processed, Heated Seed Oils

When produced responsibly, seed oils aren’t bad for you. Cold-pressed and consumed in the correct ratio for cellular health (4:1 omega-6 to omega-3), essential fatty acids from seed oils contribute to both delicious meals and good health.

The problem is that most modern diets include staggering amounts of chemically processed and rancid seed oils, which are packed into fried foods, salad dressings, baked goods, chips, packaged snacks, and more. These seed oils are processed using high heat (which turns them rancid and removes nutrients) and chemical hexane.

Seed oils are cheap to produce and often used as fillers in otherwise healthy foods. As they’ve grown in popularity, we’ve quickly started consuming them beyond healthy limits. So much exposure to omega-6 fatty acids can upset the delicate balance of omega-6 to omega-3 and trigger the inflammatory response.

2. Artificial Sweeteners

Anything that enters the gut has the ability to alter the gut microbiome — for better or worse. Highly processed foods, like artificial sweeteners, present a disadvantage since they’re not found in nature and were only recently introduced to the human diet.

One study looked at the impact of common artificial sweeteners (aspartame and sucralose) on the gut microbiome — and the results were surprising. These sugar substitutes were shown to alter gene expression in bacterial metabolism. Essentially, this turns genes “on” or “off” and can even instruct them to malfunction. Artificial sweeteners also have the ability to reduce healthy strains of bacteria and slow glucose metabolism (yikes).

3. Processed Sugar

It’s common knowledge that excess sugar consumption can negatively impact the immune system. But now that we know more about the gut microbiome, we have a better understanding of why this occurs.

Regular sugar consumption can disrupt the gut microbiome — encouraging the growth of pathogenic (unhealthy) bacteria and fungi. This can exacerbate common microbial issues like candida overgrowth. The microbiome acts as part of the immune system, with healthy bacteria regularly communicating with immune cells. So, a lack of microbiome diversity and an influx of pathogenic bacteria directly impact our immune system function.

4. Carrageenan

Carrageenan is an emulsifier commonly found in foods like yogurt, plant-based milks, baked goods, mayonnaise, sauces, and even powdered baby formula. It is used to blend water and oil without allowing separation or to “fill space” in a product, making it cheaper for the manufacturer to produce.

According to studies, though, carrageenan can have a profound impact on the gut. One stated that “clinical evidence suggests that [carrageenan] is involved in the pathogenesis and clinical management of inflammatory bowel diseases (IBD),” and it can “activate the innate immune pathways of inflammation, alter the gut microbiota composition, and decrease the thickness of the mucus barrier.” Exactly the opposite of what we want for a healthy gut. The study recommends eliminating or reducing processed foods containing carrageenan as much as possible.

5. High FODMAP Foods

Let’s make one thing clear: High FODMAP foods aren’t inherently bad. But they can aggravate gut symptoms in some people.

High FODMAP foods include some fruits, grains, lactose-containing dairy, legumes, onions, garlic, and select vegetables. The concern with high FODMAP foods is that they’re digested by the microbiome.

For people with low diversity in their gut, this could become a problem. High FODMAP foods are often triggers for people with gut dysbiosis and irritable bowel syndrome (IBS) since they ferment in the gut and can take a long time for the microbiome to digest.

If you notice that these foods are hard on your digestive system (causing symptoms like pain, gas, bloating, or diarrhea), you might benefit from a temporary low FODMAP diet.

6. Glyphosate

Glyphosate isn’t technically a food — it’s an herbicide — and it’s the most commonly used one in the world. A recent review from the Environmental Working Group found glyphosate contamination in more than ninety-five percent of oat products. But it’s also prevalent in wheat.

The problem with glyphosate is that it acts as an antifungal. So, when consumed (even at low doses but for a long period of time), it can fundamentally change the ecosystem in our gut. One study showed that more than half of the human microbiome was impacted by glyphosate exposure. The same study noted that a healthy microbiome could build up resistance to glyphosate, changing the entire gut ecosystem in the process (and not for the better).

Bonus: Some Pharmaceuticals and Other Medications

While prescription and over-the-counter medications have their place, it’s important to recognize their influence on the microbiome. Antibiotics should be used sparingly and only when truly necessary. Other popular medications like birth control, NSAIDs, SSRIs, and PPIs have all been shown to impact the fragile gut ecosystem. In a unique study, frequent use of laxatives was considered a predictor of reduced microbiome diversity in the gut.

What to Eat Instead: 6 Best Foods for Gut Health

Want to know what to eat instead of these for better gut health? Below are healthy foods help to feed the gut microbiome — welcoming a diversity of bacteria strains and promoting the good bacteria already colonized in your gut. 

1. Probiotics

Some foods contain live and active bacterial strains that can colonize the gut (enter, probiotics). Probiotic-rich foods are arguably the best way to increase your gut microbiome and diversity (yes, even better than probiotic supplements).

Probiotic Foods:

  • Yogurt
  • Kimchi
  • Kombucha
  • Some raw dairy products
  • Kefir
  • Sauerkraut

2. Amino Acids

Glycine (found in bone broth) is a powerful amino acid that helps reduce the inflammatory response in the gut. It’s incredibly soothing for people who experience regular gut dysbiosis. Other dietary amino acids, like glutamine and proline, also help create a strong gut lining.

Amino Acid Foods:

  • Grass-fed beef
  • Grass-fed organ meats
  • Eggs
  • Raw dairy
  • Bone broth
  • Collagen
  • Bone marrow

3. Resistant Starch

While resistant starch has gained a bad reputation over the years, this food is actually a powerful fuel for butyrate production. Just a word to the wise: start eating them slowly if you’re not used to resistant starch.

Resistant Starch Foods: 

  • Legumes 
  • Under-ripe bananas
  • Plantains
  • Cooked and cooled potatoes
  • Cooked and cooled rice

4. Grass-Fed Beef Liver

Nutrient deficiencies can be a huge driving factor when it comes to poor gut health. In fact, some studies have linked vitamin D deficiency to IBS. Even if a vitamin or mineral deficiency isn’t the driving force behind your gut issues, it can be helpful to add in beef liver as a super-nutrient to help replenish your body. Low nutrient absorption is likely present if you experience regular loose stools.

Easy Ways to Eat Grass-Fed Beef Liver:

  • Make a beef liver pate
  • Buy ground beef with beef liver mixed in
  • Take beef liver capsules
  • Hide it in dishes like lasagna, meatloaf, and chili

5. Raw or Low-Temperature Pasteurized Dairy

For people who are sensitive to dairy, this recommendation might seem counterintuitive. But here’s the thing: dairy is one of the most nutrient-dense foods on the planet. When unpasteurized, it contains healthy enzymes, minerals, amino acids, probiotics, almost all the fat-soluble vitamins, and protein.

Most people who struggle to digest regular dairy find raw dairy much easier on their stomachs. Dairy with A2 protein is helpful for this, too.

Raw dairy does come with some risks, and it’s important to talk to your farmer and ensure you’re getting a safe product. If you’re worried about consuming raw milk, you can try low-temperature pasteurized milk instead.

Raw Dairy Foods:

  • Milk
  • Cheese
  • Milk kefir
  • Sour cream
  • Cottage cheese
  • Yogurt

6. Farm Fresh Vegetables

It’s so important to know where your food comes from. Local farm fresh vegetables are typically easier to digest than store-bought (yes, even if it’s the same vegetable). The food transportation process usually requires the use of more chemicals for packaging and preservation.

Plus, you can ask local farmers exactly how they grow their crops. Vegetable crops that are rotated regularly to areas with fresh soil should have more vitamins and minerals in them. Plus, farms that use cover crops are less likely to deplete their soil of nutrients.

Farm Fresh Vegetable Tips:

  • Eat what’s in season
  • Eat organic vegetables
  • Ask about crop rotation
  • Ask about cover crops
  • Ask about chemicals and pesticides

Cultivate Healthy Gut Bacteria While You Change Your Diet

For people struggling with gut dysbiosis, it can be difficult to curb cravings and eat nutrient-dense foods. Yeast, parasites, and bacteria can change the way you eat and affect even the most strong-willed person.

This is why we recommend addressing your gut dysbiosis from all angles. Food, supplements, calming of the nervous system — you name it.

One of our favorite supplements for gut health is Butyrate. This is a short-chain fatty acid that’s produced in the body by a healthy gut. It does amazing things like fuel the gut lining, support blood sugar balance (promoting GLP-1), and improve brain health.

For your body to produce its own butyrate, it needs a healthy microbiome and a balanced diet that includes resistant starch. This is a tall order for those experiencing gut symptoms.

That’s what makes butyrate supplementation so powerful. You can empower your body with tools for healing while giving it the time and space required for recovery. 

Try BodyBio Butyrate

Related Articles

Ashley Palmer | 10.27.2026

Semaglutide Can Change What People Want to Eat. Here's What Your Gut Has to Do With It.

Imagine you order the meal you've loved for a decade, and a few bites in, you realize you just don't care about it. You're not full, exactly. Just uninterested.

Many people taking semaglutide (a GLP-1 medication) describe this experience. Foods that were reliable favorites suddenly seem greasy, or too sweet, or just vaguely off. Scientific American captured the strangeness: a former food writer losing interest in cooking, longtime favorites tasting wrong.

The obvious explanation is that these medications make you feel full sooner. But fullness doesn't explain why a specific curry tastes bitter now, or why a go-to wine suddenly tastes off. Something more is happening, and it points to a hormone your body has been producing your whole life.

In this article, we'll look at what the newest research suggests semaglutide does to food preferences, what GLP-1 actually does inside the body, and where your gut microbiome and a short-chain fatty acid called butyrate fit into that system.

Table of Contents:

  • Semaglutide Isn't Only Changing How Much People Eat
  • Your Gut Was Making GLP-1 Long Before Ozempic
  • Where the Microbiome Comes In
  • Can Butyrate Support Natural GLP-1 Production?
  • Why Butyrate Matters Beyond One Hormone
  • Support the System, Not Just the Signal
  • The Bigger Story Your Gut Was Already Telling

Semaglutide Isn't Only Changing How Much People Eat

Researchers are starting to document what users have been describing anecdotally.

A 2026 observational study of hospitalized Japanese patients with type 2 diabetes found that those on semaglutide showed significantly reduced preference for sweet carbohydrates, non-sweet carbohydrates, and non-sweet fats — while preferences for protein and dietary fiber stayed essentially unchanged. This wasn't a blanket loss of interest in food. It was a selective shift.

For someone focused on weight loss, eating less may be the point. Still, a reduced appetite for fats is worth paying attention to, since dietary fats supply essential fatty acids and help the body absorb fat-soluble vitamins.

Taste perception may also contribute. In a 16-week, randomized, placebo-controlled study of 30 women with obesity and PCOS, semaglutide improved overall taste recognition scores and altered brain activity in response to both food images and a sweet solution. In other words, participants' sense of taste sharpened, which may help explain why some foods suddenly seem too strong.

A 2025 study found the opposite: reduced sensitivity across all five basic tastes in people taking GLP-1 medications compared with matched non-users. That means the more accurate takeaway isn't "Ozempic makes food taste different" so much as "GLP-1 signaling reaches further into how we experience food than many researchers expected."

None of these studies settles the question on its own. But together they point somewhere useful: GLP-1 isn't only a satiety signal. It appears to play a role in how appealing food seems in the first place.

Your Gut Was Making GLP-1 Long Before Ozempic

Here's what often gets lost: GLP-1 wasn't invented by a pharmaceutical company.

GLP-1 is a hormone your intestines release on their own, produced mainly by specialized cells called enteroendocrine L-cells. These cells sit in the gut lining, acting as sensors that detect what's arriving from your meal and releasing hormones that coordinate the response.

GLP-1 helps regulate insulin release, slows how quickly the stomach empties, contributes to satiety, and carries information from the gut to the brain. GLP-1 medications didn't create a new pathway. They target one your body was already running.

But there's an important difference, and a lot of wellness marketing glosses over it. Your body's own GLP-1 is broken down within minutes of release. Semaglutide is engineered to resist that breakdown and activate GLP-1 receptors far more strongly, for far longer.

Supporting your body's own GLP-1 signaling through food or the microbiome isn't a lesser version of taking semaglutide. It's a different thing entirely.

Where the Microbiome Comes In

Your gut bacteria eat what you don't digest.

When fermentable fibers and resistant starch reach the colon, bacteria ferment them and produce short-chain fatty acids (SCFAs), mainly acetate, propionate, and butyrate. These aren't waste products. They're signaling molecules, and the cells lining your intestines are built to receive them.

Intestinal cells carry receptors, including FFAR2 (also called GPR43) and FFAR3, that detect short-chain fatty acids. When SCFAs bind these receptors on L-cells, they trigger an internal cascade that can influence the release of gut hormones, including GLP-1 and a related satiety hormone called PYY.

So the pathway is real: fiber arrives, microbes ferment it, metabolites are produced, gut cells sense them, hormones get released, and the gut communicates with the pancreas and the brain. That last link is worth sitting with — your gut and brain are in constant conversation, and food-derived microbial metabolites are part of what they're saying to each other.

The quick version of this compresses to: fiber → butyrate → GLP-1 → less hunger. The actual biology is an ecosystem with many signals, of which butyrate is one.

Can Butyrate Support Natural GLP-1 Production?

The mechanistic evidence is strong. The human evidence is thinner, but still developing.

A small 2026 study in adults with obesity found that oral sodium butyrate combined with L-arginine significantly increased circulating GLP-1. That's encouraging, but the study had only seven participants, butyrate wasn't tested on its own, and participants reported no difference in hunger or fullness.

By contrast, in a 2025 randomized controlled trial of 50 adults with obesity, people taking sodium butyrate alongside a reduced-calorie diet saw reductions in weight, BMI, and waist circumference, but their serum GLP-1 didn't change significantly.

A 2026 randomized, double-blind trial of 46 adults added more nuance. After 12 weeks of sodium butyrate plus a moderately reduced-calorie diet, participants with obesity lost a meaningful amount of weight. Participants who also had type 2 diabetes did not, although their blood sugar control improved.

That pattern echoes an earlier finding. In a 2018 study, four weeks of oral butyrate improved insulin sensitivity in lean men but not in men with metabolic syndrome.

Together, these suggest butyrate's effects depend heavily on metabolic context: who you are, what your microbiome looks like, and where the butyrate ends up. But GLP-1 is only part of the story.

Why Butyrate Matters Beyond One Hormone

Focusing only on GLP-1 undersells what butyrate does.

Butyrate is the primary fuel source for colonocytes, the cells lining your colon. Not a supporting nutrient. Their main energy supply. Those cells use butyrate to power the work of maintaining a functioning gut lining, the barrier that decides what passes from your digestive tract into your body and what doesn't.

This is where the cellular health picture comes together. A well-fueled intestinal lining supports a healthy gut barrier. A healthy barrier supports a stable environment for the microbial community living there. And that community produces metabolites that signal to the rest of the body — including the gut-to-brain communication involved in appetite and satiety.

Butyrate sits at the center of that loop as both fuel and message. That role holds regardless of what the GLP-1 research eventually concludes.

Support the System, Not Just the Signal

The takeaway isn't that you should be trying to maximize GLP-1. Your metabolism and gut function are more complex than one signal.

Lasting metabolic regulation runs on an ecosystem: microbial diversity, adequate fermentable fiber, well-nourished intestinal cells, an intact gut barrier, and steady communication between gut and brain. Semaglutide shows what happens when you push one signal in that system very hard. It doesn't show that one signal is the whole system, or the only way to modulate it.

Feeding that ecosystem starts with fiber-rich whole foods: vegetables, legumes, cooked-and-cooled potatoes and rice (good sources of resistant starch), and other plant foods your gut bacteria can thrive on. Give it time. Gut environments shift over weeks and months, not days.

For targeted support, BodyBio Butyrate provides butyrate in supplement form to support the gut lining and a healthy microbiome.*

The Bigger Story Your Gut Was Already Telling

Semaglutide has taught researchers a great deal about how strongly GLP-1 signaling can influence not just hunger but desires: what sounds good, what tastes right, and what gets left on the plate.

And yet, the more interesting revelation may be how much was already happening without it. Every meal you eat feeds a microbial community that turns fiber into signaling molecules, which intestinal cells read and translate into messages sent onward to your pancreas and your brain. That conversation has been running your entire life.

Butyrate is one voice in that conversation. It fuels the cells that hold your gut barrier together and takes part in the signaling between your microbiome and the rest of your body. Supporting butyrate production in the gut won't replicate a pharmaceutical. It doesn't need to. Feeding the system well is its own goal, and the benefits build gradually, often in small shifts that are easy to miss at first.

Support your gut at the cellular level with BodyBio Butyrate.*

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to diagnose, treat, cure, or prevent any disease.

This article is for educational purposes only and is not a substitute for medical advice. If you're taking semaglutide or another GLP-1 medication, talk with your healthcare provider before adding new supplements.

Ashley Palmer | 06.16.2026

Comprehensive Guide to Butyrate: What it is, Benefits, Side Effects, & More

Butyrate is a necessary component to a balanced microbiome, also working as an inflammation guard*. One of several, short-chain fatty acids created from fermented resistant starches, low butyrate levels have been associated with serious health concerns.

Ashley Palmer | 04.24.2026

Butyrate and IBS: What Your Gut Cells Actually Need

You've tried the probiotics. You've cleaned up your diet. You're doing everything you're supposed to do, and your gut still isn't cooperating.

If you have IBS, that frustration is familiar. Bloating, urgency, unpredictable mornings, the constant guessing game of what's going to set things off.

What often gets missed in the conversation around IBS isn't a trendy new supplement or a stricter elimination diet. It's something more foundational: what your gut's own cells actually need to function.

Here's a closer look at what butyrate does, why IBS and low butyrate levels are closely linked, and how supporting the gut at the cellular level can make a difference.

Table of Contents:

  • Understanding Butyrate

  • How Butyrate Works in the Gut

  • Why This Becomes an Issue for People With IBS

  • Supporting the Body More Effectively

  • Butyrate, IBS, and the Cellular Health Connection

  • How Butyrate Supports IBS Comfort Long-Term

Understanding Butyrate

Butyrate is a short-chain fatty acid (SCFA) produced in the colon when beneficial gut bacteria ferment dietary fiber, specifically resistant starch. It belongs to a class of compounds called postbiotics, the functional byproducts of a healthy microbiome.

Unlike probiotics, which are living bacteria, butyrate is a metabolite. It's a compound your body puts to work directly at the cellular level. Understanding the difference between probiotics and postbiotics, like butyrate, is a useful shift you can make when thinking about gut health.

Why the Body Relies on Butyrate

Your colon is lined with specialized cells called colonocytes. These cells run almost entirely on butyrate, providing up to 90% of their energy needs.

When colonocytes have what they need, they do their job well. They maintain the integrity of the gut lining, regulate what passes into the bloodstream, support a healthy inflammatory response, and help keep gut motility on track. Without enough butyrate, the gut’s main source of energy, the gut simply can't do its job well.

How Butyrate Works in the Gut

Butyrate supports three interconnected systems in the gut: the gut lining, the immune environment, and gut motility.

The gut lining is just one cell layer thick. Butyrate fuels those cells and supports the tight junctions between them, the structural connections that keep the barrier intact and functioning.

At the immune level, butyrate helps the body maintain a balanced inflammatory response in the colon without triggering overactivation. And because it directly influences the rhythmic contractions that move contents through the digestive tract, it plays a meaningful role in the irregular patterns that so many people with IBS experience.

What Happens When Butyrate Levels Are Supported vs. Strained

When butyrate is available in adequate amounts, the gut lining stays resilient, motility is more regular, and digestive comfort improves, whether you have a diagnosis of IBS or not.

If butyrate levels fall short, the gut barrier may become less stable, motility can become unpredictable, and the colon's immune environment may shift. How pronounced these patterns are varies from person to person. Diet, stress, genetics, and microbiome composition all play a role in your body’s patterns too.

Why This Becomes an Issue for People With IBS

Modern Stressors on Butyrate Production

Butyrate is made by gut bacteria that ferment resistant starch, a type of fiber found in foods like cooked and cooled potatoes, legumes, and underripe bananas. The modern diet is low in these foods. That means many people simply aren't giving their gut bacteria the raw materials they need to produce adequate butyrate levels.

Antibiotic use, chronic stress, and certain medications can also reduce the population of butyrate-producing bacteria in the colon. Over time, that compounds the butyrate production gap.

Why Common Approaches Often Fall Short

Probiotics can be a valuable part of gut health support, but most probiotic strains are not butyrate-producing species. They can shift the microbiome, but they don't directly address the fuel shortage that many IBS-related symptoms may stem from.

Fiber-based approaches come with a similar challenge. In some people with IBS, increasing fermentable fiber can worsen bloating and gas before it helps, because a disrupted microbiome may not efficiently use that fiber to make butyrate.

Since butyrate works at the cellular level, the signs of low butyrate can overlap common IBS symptoms, which is part of why the connection between the two gets overlooked.

Supporting the Body More Effectively

Foundational Support for Butyrate Production

Diet is the first step to improving the body's natural butyrate production. Foods high in resistant starch give butyrate-producing bacteria what they need to function properly.

Foods that naturally support butyrate production include cooked and cooled rice, potatoes, and legumes. A steady intake of resistant starch over time does more for the microbiome than occasional high-fiber days followed by low-fiber ones.

Stress management and adequate sleep can also support a more stable gut environment. Chronic stress directly affects microbiome composition and motility through the gut-brain axis. 

When Targeted Butyrate Support Makes Sense

For people whose microbiome is disrupted or whose diet can't consistently provide enough resistant starch, direct butyrate supplementation is another option for long-term support.*

Supplemental butyrate delivers the short-chain fatty acid directly to the colon, where colonocytes can put it to use.* Clinical research has shown that sodium butyrate supplementation is associated with improvements in abdominal comfort and bowel regularity in people with IBS.

Sodium butyrate and calcium magnesium butyrate are both effective options, and the differences between which works best for your body often come down to your individual health history and mineral needs.*

Butyrate, IBS, and the Cellular Health Connection

IBS is complex, and its causes vary from person to person. But one consistent finding in the research is that people with IBS tend to have lower levels of butyrate-producing bacteria and overall reduced short-chain fatty acids in the gut. That points to a cellular resource problem as much as a microbiome problem.

When the cells lining the colon are undersupported, the entire digestive environment becomes less stable. Gut barrier function, motility, immune signaling, and communication along the gut-brain axis all depend on colonocytes having what they need to function.

Supporting the gut at the cellular level is not a replacement for other gut health practices (whole food probiotics, exercise, etc.). It’s the foundation that makes those practices more effective. When your cells are supported, the body functions more efficiently as a whole.

How Butyrate Supports IBS Comfort Long-Term

IBS can feel like a moving target, and the path toward better digestive comfort is rarely a straight line or a quick fix. Supporting your gut at the cellular level creates a more stable foundation, and that starts with making sure colonocytes have the fuel they need.*

Butyrate works best as part of a consistent approach that includes diet, lifestyle, and targeted support where needed. Progress tends to be gradual, and that's expected. It reflects the time it takes for the gut lining to strengthen and the microbiome to rebalance.

IBS makes a lot more sense when you know what the gut's cells actually need. And that clarity is often where real progress begins.

Support your gut at the cellular level with BodyBio Butyrate.*